- abnormal rib morphology / MGI
- abnormal sternum morphology / MGI
- decreased bone marrow cell number / MGI
- kinked tail / MGI
- abnormal branching of the mammary ductal tree / MGI
- spleen hypoplasia / MGI
- open neural tube / MGI
- small ovary / MGI
- small testis / MGI
- abnormal epidermal layer morphology / MGI
- abnormal epidermis stratum basale morphology / MGI
- abnormal epidermis stratum spinosum morphology / MGI
- absent epidermis stratum corneum / MGI
- decreased body weight / MGI
- impaired hematopoiesis / MGI
- abnormal embryo development / MGI
- decreased embryo size / MGI
- abnormal visceral yolk sac morphology / MGI
- male infertility / MGI
- female infertility / MGI
- neoplasm / MGI
- increased tumor incidence / MGI
- increased T cell derived lymphoma incidence / MGI
- increased carcinoma incidence / MGI
- decreased tumor incidence / MGI
- abnormal skin morphology / MGI
- abnormal motor capabilities/coordination/movement / MGI
- premature death / MGI
- abnormal developmental patterning / MGI
- abnormal embryonic tissue morphology / MGI
- abnormal extraembryonic tissue morphology / MGI
- abnormal axial skeleton morphology / MGI
- no abnormal phenotype detected / MGI
- abnormal hematopoietic system morphology/development / MGI
- absent ovarian follicles / MGI
- abnormal chromosome morphology / MGI
- embryonic growth retardation / MGI
- increased mortality induced by ionizing radiation / MGI
- abnormal mitosis / MGI
- fetal growth retardation / MGI
- decreased incidence of tumors by chemical induction / MGI
- enlarged allantois / MGI
- absent germ cells / MGI
- increased thymus weight / MGI
- abnormal amnion morphology / MGI
- sepsis / MGI
- azoospermia / MGI
- growth/size/body region phenotype / MGI
- reproductive system phenotype / MGI
- abnormal cell physiology / MGI
- increased apoptosis / MGI
- early cellular replicative senescence / MGI
- abnormal DNA repair / MGI
- decreased survivor rate / MGI
- decreased common myeloid progenitor cell number / MGI
- chromosomal instability / MGI
- increased sensitivity to induced cell death / MGI
- adrenal gland hyperplasia / MGI
- decreased fetal weight / MGI
- decreased birth weight / MGI
- increased mammary gland tumor incidence / MGI
- decreased tumor latency / MGI
- abnormal double-strand DNA break repair / MGI
- postnatal lethality, incomplete penetrance / MGI
- neonatal lethality, incomplete penetrance / MGI
- perinatal lethality, incomplete penetrance / MGI
- prenatal lethality, complete penetrance / MGI
- embryonic lethality, complete penetrance / MGI
- embryonic lethality between somite formation and embryo turning, complete penetrance / MGI
- embryonic lethality during organogenesis, complete penetrance / MGI
- prenatal lethality, incomplete penetrance / MGI
- embryonic lethality during organogenesis, incomplete penetrance / MGI
- lethality throughout fetal growth and development, incomplete penetrance / MGI
- abnormal proamniotic cavity morphology / MGI
- decreased fibroblast proliferation / MGI
- increased lymphoma incidence / MGI
- ovary degeneration / MGI
B6.129P2-Brca2tm2Mak/Cnbc
Status | Available to order |
EMMA ID | EM:06747 |
International strain name | B6.129P2-Brca2tm2Mak/Cnbc |
Alternative name | BrCa2 |
Strain type | Targeted Mutant Strains : Conditional mutation |
Allele/Transgene symbol | Brca2tm2Mak |
Gene/Transgene symbol | Brca2 |
Information from provider
Provider | Tak W Mak |
Provider affiliation | Mak Lab, Campbell Family Breast Cancer reseach Institute |
Genetic information | Conditional targeted mutation of exons 9 and 10 flanked by loxP sites. |
Phenotypic information | Brca2-null mice have a defect in embryonic cellular proliferation and die in utero. tBrca2/ T cells were more likely than wild-type cells to undergo spontaneous apoptosis, but apoptosed normally in response to restimulation or DNA-damaging stress signals. |
Breeding history | Backcrossed 10 times to C57BL/6. |
References |
|
Homozygous fertile | yes |
Homozygous viable | yes |
Homozygous matings required | no |
Immunocompromised | no |
Information from EMMA
Archiving centre | CNB-CSIC, Centro Nacional de Biotecnologia, Madrid, Spain |
Animals used for archiving | homozygous C57BL/6J, wild-type C57BL/6J |
Stage of embryos | 2-cell |
Disease and phenotype information
Orphanet associated rare diseases, based on orthologous gene matching
- Hereditary breast and ovarian cancer syndrome / Orphanet_145
- Fanconi anemia / Orphanet_84
- Inherited cancer-predisposing syndrome due to biallelic BRCA2 mutations / Orphanet_319462
- Cholangiocarcinoma / Orphanet_70567
MGI phenotypes (gene matching)
Literature references
- Loss of Brca2 and p53 synergistically promotes genomic instability and deregulation of T-cell apoptosis.;Cheung Alison M Y, Hande M Prakash, Jalali Farid, Tsao Ming-Sound, Skinnider Brian, Hirao Atsushi, McPherson J Peter, Karaskova Jana, Suzuki Akira, Wakeham Andrew, You-Ten Annick, Elia Andrew, Squire Jeremy, Bristow Rob, Hakem Razqallah, Mak Tak W, ;2002;Cancer research;62;6194-204; 12414647
- Brca2 deficiency does not impair mammary epithelium development but promotes mammary adenocarcinoma formation in p53(+/-) mutant mice.;Cheung Alison M Y, Elia Andrew, Tsao Ming-Sound, Done Susan, Wagner Kay-Uwe, Hennighausen Lothar, Hakem Razqallah, Mak Tak W, ;2004;Cancer research;64;1959-65; 15026330
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