- hyperactivity / MGI
- impaired coordination / MGI
- abnormal dopamine level / MGI
- abnormal pain threshold / MGI
- abnormal noradrenaline level / MGI
- impaired conditioned place preference behavior / MGI
- impaired behavioral response to addictive substance / MGI
- impaired behavioral response to morphine / MGI
- abnormal nervous system physiology / MGI
- abnormal thermosensation / MGI
- decreased inflammatory response / MGI
- increased airway responsiveness / MGI
- abnormal touch/ nociception / MGI
- abnormal respiratory mucosa goblet cell morphology / MGI
- abnormal chemical nociception / MGI
- joint inflammation / MGI
- decreased nerve fiber response intensity / MGI
- hyperalgesia / MGI
- abnormal pulmonary respiratory rate / MGI
- decreased aggression / MGI
B6.129P2-Tacr1tm1Sph/H
Status | Available to order |
EMMA ID | EM:05452 |
International strain name | B6.129P2-Tacr1tm1Sph/H |
Alternative name | NK1 Receptor knock-out / Backcrossed on C57BL/6 |
Strain type | Targeted Mutant Strains : Knock-out |
Allele/Transgene symbol | Tacr1tm1Sph |
Gene/Transgene symbol | Tacr1 |
Information from provider
Provider | S P Hunt |
Provider affiliation | Windeyer Institute of Medical Sciences, University College London |
Genetic information | Targeting construct for NK1 (or Tacr1) receptor is derived from 129/Sv DNA. A reporter/selection cassette (IRES-Gal-MC1-Neo) was inserted into exon 1 of the targeted allele, rendering the mRNA out-of-frame and thus no functional NK1 receptor can be produced. |
Phenotypic information | Deletion of Tacr1/NK1 receptor gene. Viable, normal breeding pattern. No obvious phenotype. |
Breeding history | Backcrossed for more than 10 generations. |
References |
|
Homozygous fertile | yes |
Homozygous viable | yes |
Homozygous matings required | no |
Immunocompromised | no |
Information from EMMA
Archiving centre | Mary Lyon Centre at MRC Harwell, Oxford, United Kingdom |
Animals used for archiving | heterozygous C57BL/6 |
Breeding at archiving centre | Males were archived upon arrival at the archiving centre. No breeding was carried out. |
Disease and phenotype information
MGI allele-associated human disease models
MGI phenotypes (allele matching)
MGI phenotypes (gene matching)
- decreased anxiety-related response / MGI
- hyperactivity / MGI
- impaired coordination / MGI
- abnormal body temperature homeostasis / MGI
- abnormal humoral immune response / MGI
- decreased inflammatory response / MGI
- abnormal dopamine level / MGI
- increased airway responsiveness / MGI
- abnormal touch/ nociception / MGI
- abnormal pain threshold / MGI
- abnormal nervous system electrophysiology / MGI
- abnormal respiratory mucosa goblet cell morphology / MGI
- abnormal mast cell physiology / MGI
- decreased circulating corticosterone level / MGI
- abnormal chemical nociception / MGI
- abnormal nociception after inflammation / MGI
- joint inflammation / MGI
- decreased vascular permeability / MGI
- decreased nerve fiber response intensity / MGI
- abnormal nervous system physiology / MGI
- abnormal thermosensation / MGI
- abnormal noradrenaline level / MGI
- abnormal neuron physiology / MGI
- hyperalgesia / MGI
- hyporesponsive to tactile stimuli / MGI
- decreased aggression / MGI
- increased chemical nociceptive threshold / MGI
- abnormal physiological response to xenobiotic / MGI
- impaired conditioned place preference behavior / MGI
- impaired behavioral response to xenobiotic / MGI
- impaired behavioral response to addictive substance / MGI
- impaired behavioral response to morphine / MGI
- decreased vocalization / MGI
- abnormal pulmonary respiratory rate response / MGI
Literature references
- Altered nociception, analgesia and aggression in mice lacking the receptor for substance P.;De Felipe C, Herrero J F, O'Brien J A, Palmer J A, Doyle C A, Smith A J, Laird J M, Belmonte C, Cervero F, Hunt S P, ;1998;Nature;392;394-7; 9537323
- Genetic background influences the behavioural and molecular consequences of neurokinin-1 receptor knockout.;McCutcheon J E, Fisher A S, Guzdar E, Wood S A, Lightman S L, Hunt S P, ;2008;The European journal of neuroscience;27;683-90; 18279320
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