- abnormal cell morphology / MGI
- abnormal crypts of Lieberkuhn morphology / MGI
- abnormal intestinal mucosa morphology / MGI
- increased body weight / MGI
- hyperglycemia / MGI
- neoplasm / MGI
- increased colonic adenoma incidence / MGI
- increased circulating insulin level / MGI
- hepatic steatosis / MGI
- increased liver weight / MGI
- large intestinal inflammation / MGI
- oxidative stress / MGI
- increased incidence of tumors by chemical induction / MGI
- impaired glucose tolerance / MGI
- insulin resistance / MGI
- increased susceptibility to weight gain / MGI
- increased susceptibility to diet-induced obesity / MGI
- abnormal mitochondrion morphology / MGI
- abnormal mitochondrial physiology / MGI
- abnormal epididymal fat pad morphology / MGI
- increased susceptibility to induced colitis / MGI
- abnormal renal fat pad morphology / MGI
- increased sensitivity to xenobiotic induced morbidity/mortality / MGI
- abnormal respiratory electron transport chain / MGI
- abnormal oxidative phosphorylation / MGI
- increased mitochondria number / MGI
- abnormal mitophagy / MGI
- increased body fat mass / MGI
- increased susceptibility to diet-induced non-insulin dependent diabetes / MGI
B6.129-Trp53inp1tm1Acar/Orl
Status | Available to order |
EMMA ID | EM:02038 |
International strain name | B6.129-Trp53inp1tm1Acar/Orl |
Alternative name | TP53INP1 KO B6 |
Strain type | Targeted Mutant Strains : Knock-out |
Allele/Transgene symbol | Trp53inp1tm1Acar |
Gene/Transgene symbol | Trp53inp1 |
Information from provider
Provider | Alice Carrier |
Provider affiliation | Inserm U624 |
Genetic information | A null mutation was introduced in 129/Sv ES cells by replacing coding exons 2, 3 and 4 of Trp53inp1 gene with a neomycin resistance cassette. This cassette contains the neo gene and the gene encoding the cre recombinase designed for self-excision in male germline, flanked by loxP sites (Bunting M et al., Genes&Dev. 1999, 13/1524; Genbank AF169416). |
Phenotypic information | None. |
Breeding history | Mutation was introduced in 129/Sv ES cells, chimeric males were mated to C57BL/6 females to generate heterozygous pups on mixed 129/Sv x C57BL/6 background, then backcrossed on C57BL/6 background for 8 generations. The strain is currently bred on B6 background. |
References |
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Information from EMMA
Archiving centre | Institut de Transgenose, INTRAGENE, Orléans, France |
Disease and phenotype information
MGI allele-associated human disease models
MGI phenotypes (allele matching)
MGI phenotypes (gene matching)
- abnormal cell morphology / MGI
- abnormal crypts of Lieberkuhn morphology / MGI
- abnormal intestinal mucosa morphology / MGI
- increased body weight / MGI
- hyperglycemia / MGI
- neoplasm / MGI
- increased colon adenoma incidence / MGI
- increased circulating insulin level / MGI
- hepatic steatosis / MGI
- increased liver weight / MGI
- large intestinal inflammation / MGI
- oxidative stress / MGI
- increased incidence of tumors by chemical induction / MGI
- impaired glucose tolerance / MGI
- insulin resistance / MGI
- increased susceptibility to weight gain / MGI
- increased susceptibility to diet-induced obesity / MGI
- abnormal mitochondrion morphology / MGI
- abnormal mitochondrial physiology / MGI
- abnormal epididymal fat pad morphology / MGI
- increased susceptibility to induced colitis / MGI
- abnormal renal fat pad morphology / MGI
- increased sensitivity to xenobiotic induced morbidity/mortality / MGI
- abnormal respiratory electron transport chain / MGI
- abnormal oxidative phosphorylation / MGI
- increased mitochondria number / MGI
- abnormal mitophagy / MGI
- increased body fat mass / MGI
- increased susceptibility to diet-induced non-insulin dependent diabetes / MGI
Literature references
- Molecular and functional characterization of the stress-induced protein (SIP) gene and its two transcripts generated by alternative splicing. SIP induced by stress and promotes cell death.;Tomasini R, Samir A A, Vaccaro M I, Pebusque M J, Dagorn J C, Iovanna J L, Dusetti N J, ;2001;The Journal of biological chemistry;276;44185-92; 11557757
- p53DINP1, a p53-inducible gene, regulates p53-dependent apoptosis.;Okamura S, Arakawa H, Tanaka T, Nakanishi H, Ng C C, Taya Y, Monden M, Nakamura Y, ;2001;Molecular cell;8;85-94; 11511362
- P53-dependent expression of the stress-induced protein (SIP).;Tomasini Richard, Samir Amina Azizi, Pebusque Marie-Josèphe, Calvo Ezequiel L, Totaro Serena, Dagorn Jean Charles, Dusetti Nelson J, Iovanna Juan L, ;2002;European journal of cell biology;81;294-301; 12067065
- TP53INP1s and homeodomain-interacting protein kinase-2 (HIPK2) are partners in regulating p53 activity.;Tomasini Richard, Samir Amina Azizi, Carrier Alice, Isnardon Daniel, Cecchinelli Barbara, Soddu Silvia, Malissen Bernard, Dagorn Jean-Charles, Iovanna Juan L, Dusetti Nelson J, ;2003;The Journal of biological chemistry;278;37722-9; 12851404
- Down-expression of tumor protein p53-induced nuclear protein 1 in human gastric cancer.;Jiang Pei-Hong, Motoo Yoshiharu, Garcia Stéphane, Iovanna Juan Lucio, Pébusque Marie-Josèphe, Sawabu Norio, ;2006;World journal of gastroenterology;12;691-6; 16521180
- Colitis and colitis-associated cancer are exacerbated in mice deficient for tumor protein 53-induced nuclear protein 1.;Gommeaux Julien, Cano Carla, Garcia Stéphane, Gironella Meritxell, Pietri Sylvia, Culcasi Marcel, Pébusque Marie-Josèphe, Malissen Bernard, Dusetti Nelson, Iovanna Juan, Carrier Alice, ;2007;Molecular and cellular biology;27;2215-28; 17242209
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