- decreased susceptibility to autoimmune diabetes / MGI
- periinsulitis / MGI
- decreased physiological sensitivity to xenobiotic / MGI
- decreased hematocrit / MGI
- abnormal liver physiology / MGI
- abnormal T cell differentiation / MGI
- abnormal cytokine secretion / MGI
- increased autoantibody level / MGI
- decreased susceptibility to parasitic infection / MGI
NOD.B6-(D17Mit16-D17Mit36)/GhjCnrm
Status | Available to order |
EMMA ID | EM:01398 |
International strain name | NOD.B6-(D17Mit16-D17Mit36)/GhjCnrm |
Alternative name | NOD.B6-C17(R2) |
Strain type | Other |
Allele/Transgene symbol | Idd1C57BL/6, H2b |
Gene/Transgene symbol | Idd1, H2 |
Information from provider
Provider | Henri-Jean Garchon |
Provider affiliation | ER70 CNRS et Unite 125 Inserm |
Genetic information | NOD congenic strain with the H2 complex from C57BL/6. The R2 interval includes the Idd1 but excludes the Idd16 locus. |
Phenotypic information | This line and the related lines, consomic, congenic and subcongenic for chromosome 17 and fragments of this chromosome, were derived to thoroughly investigate the role of the major histocompatibility complex (MHC) and MHC-linked genes in autoimmune predisposition, notably including type I diabetes (Idd1 and Idd16) loci and chronic autoimmune thyroiditis (Ceat1) locus, on the non-obese diabetic (NOD) background. The R2 line is completely protected against type 1 diabetes. |
Breeding history | 20 generations of backcrosses followed by sister-brother matings. Selection of subcongenics by additional backcrosses. |
References |
|
Information from EMMA
Archiving centre | CNR, Consiglio Nazionale delle Ricerche, Monterotondo, Italy |
Disease and phenotype information
MGI phenotypes (allele matching)
MGI phenotypes (gene matching)
- decreased hematocrit / MGI
- abnormal liver physiology / MGI
- abnormal T cell differentiation / MGI
- increased susceptibility to bacterial infection / MGI
- abnormal immunoglobulin level / MGI
- increased IgM level / MGI
- abnormal cytokine secretion / MGI
- thyroid inflammation / MGI
- increased autoantibody level / MGI
- insulitis / MGI
- increased susceptibility to autoimmune diabetes / MGI
- decreased susceptibility to autoimmune diabetes / MGI
- decreased susceptibility to parasitic infection / MGI
- immune system phenotype / MGI
- periinsulitis / MGI
- abnormal T cell number / MGI
- decreased CD4-positive, alpha beta T cell number / MGI
- increased CD8-positive, alpha-beta T cell number / MGI
- decreased IgG1 level / MGI
- decreased IgG2a level / MGI
- increased physiological sensitivity to xenobiotic / MGI
- decreased physiological sensitivity to xenobiotic / MGI
- increased sensitivity to induced morbidity/mortality / MGI
- abnormal glucose homeostasis / MGI
- periinsulitis / MGI
- increased CD4-positive, alpha beta T cell number / MGI
Literature references
- An interval tightly linked to but distinct from the H2 complex controls both overt diabetes (Idd16) and chronic experimental autoimmune thyroiditis (Ceat1) in nonobese diabetic mice.;Boulard Olivier, Damotte Diane, Deruytter Nathalie, Fluteau Guy, Carnaud Claude, Garchon Henri-Jean, ;2002;Diabetes;51;2141-7; 12086944
- Mapping non-class II H2-linked loci for type 1 diabetes in nonobese diabetic mice.;Deruytter Nathalie, Boulard Olivier, Garchon Henri-Jean, ;2004;Diabetes;53;3323-7; 15561967
Information on how we integrate external resources can be found here
INFRAFRONTIER® and European Mouse Mutant Archive - EMMA® are registered trademarks at the European Union Intellectual Property Office (EUIPO).