- abnormal heart morphology / IMPC
- abnormal behavior / IMPC
- decreased thigmotaxis / IMPC
- enlarged spleen / IMPC
- abnormal spleen morphology / IMPC
- abnormal lymph node morphology / IMPC
- enlarged lymph nodes / IMPC
- abnormal eye morphology / IMPC
- hyperplasia / IMPC
- decreased anxiety-related response / IMPC
- small kidney / IMPC
- abnormal kidney morphology / IMPC
- increased heart atrium size / IMPC
- abnormal skin morphology / IMPC
- abnormal lung morphology / IMPC
- single kidney / IMPC
- enlarged kidney / IMPC
- decreased locomotor activity / IMPC
B10.Cg-Ncf1m1J H2q/Kctt
Information from provider
Provider | Rikard Holmdahl |
Provider affiliation | Lund University, CMB, Section for medical inflammation reseach |
Genetic information | The strain lacks expression of Ncf1 when homozygous for the mutation. The strain is based on The Jackson Laboratory's strain B6.Cg-Dock7m +/+ Leprdb/J (JAX stock ID 000697). However, this strain lacks the Lepr mutation, and basically only the Ncf1 gene remains from the original strain (minimal fragment). It is also backcrossed to C57BL/10.Q to a pure background (speed congenics). |
Phenotypic information | None. |
Breeding history | Backcrossed to B10.Q for more than 10 generations by speed congenics. |
References |
|
Information from EMMA
Archiving centre | Karolinska Institutet, Stockholm, Sweden |
Stage of embryos | 2-cell |
Disease and phenotype information
MGI allele-associated human disease models
Orphanet associated rare diseases, based on orthologous gene matching
- Chronic granulomatous disease / Orphanet_379
IMPC phenotypes (gene matching)
MGI phenotypes (gene matching)
- decreased hematocrit / MGI
- abnormal liver physiology / MGI
- abnormal T cell differentiation / MGI
- increased susceptibility to bacterial infection / MGI
- abnormal immunoglobulin level / MGI
- increased IgM level / MGI
- abnormal cytokine secretion / MGI
- thyroid inflammation / MGI
- increased autoantibody level / MGI
- insulitis / MGI
- increased susceptibility to autoimmune diabetes / MGI
- decreased susceptibility to autoimmune diabetes / MGI
- decreased susceptibility to parasitic infection / MGI
- immune system phenotype / MGI
- periinsulitis / MGI
- abnormal T cell number / MGI
- decreased CD4-positive, alpha beta T cell number / MGI
- increased CD8-positive, alpha-beta T cell number / MGI
- decreased IgG1 level / MGI
- decreased IgG2a level / MGI
- increased physiological sensitivity to xenobiotic / MGI
- decreased physiological sensitivity to xenobiotic / MGI
- increased sensitivity to induced morbidity/mortality / MGI
- abnormal angiogenesis / MGI
- abnormal heart morphology / MGI
- altered response to myocardial infarction / MGI
- abnormal lung morphology / MGI
- skin lesions / MGI
- abnormal spatial learning / MGI
- reduced long term potentiation / MGI
- abnormal cardiovascular system physiology / MGI
- cardiac hypertrophy / MGI
- abnormal postnatal growth / MGI
- abnormal inflammatory response / MGI
- abnormal respiratory system physiology / MGI
- abnormal tracheal smooth muscle morphology / MGI
- increased susceptibility to infection / MGI
- decreased susceptibility to viral infection / MGI
- abnormal macrophage physiology / MGI
- abnormal neutrophil physiology / MGI
- granulomatous inflammation / MGI
- dilated heart left ventricle / MGI
- increased systemic arterial blood pressure / MGI
- abnormal bile duct morphology / MGI
- abnormal cardiac muscle contractility / MGI
- enhanced long term potentiation / MGI
- decreased vasoconstriction / MGI
- increased angiotensin I-converting enzyme activity / MGI
- abnormal locomotor activation / MGI
- liver abscess / MGI
- increased circulating renin level / MGI
- abnormal intestinal goblet cell morphology / MGI
- oxidative stress / MGI
- abnormal vascular endothelial cell physiology / MGI
- abnormal vascular wound healing / MGI
- abnormal response to infection / MGI
- impaired granulocyte bactericidal activity / MGI
- increased acute inflammation / MGI
- immune system phenotype / MGI
- abnormal vascular smooth muscle physiology / MGI
- impaired cued conditioning behavior / MGI
- increased sensitivity to induced morbidity/mortality / MGI
- increased lung compliance / MGI
- decreased airway resistance / MGI
Literature references
- Enhanced autoimmunity, arthritis, and encephalomyelitis in mice with a reduced oxidative burst due to a mutation in the Ncf1 gene.;Hultqvist Malin, Olofsson Peter, Holmberg Jens, Bäckström B Thomas, Tordsson Jesper, Holmdahl Rikard, ;2004;Proceedings of the National Academy of Sciences of the United States of America;101;12646-51; 15310853
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