- absent kidney / MGI
- abnormal kidney development / MGI
- delayed kidney development / MGI
- decreased thymocyte number / MGI
- abnormal ovary morphology / MGI
- abnormal oogenesis / MGI
- secondary sex reversal / MGI
- no abnormal phenotype detected / MGI
- abnormal secondary sex determination / MGI
- abnormal adrenal gland physiology / MGI
- no phenotypic analysis / MGI
- kidney failure / MGI
- nervous system phenotype / MGI
- abnormal reproductive system development / MGI
- abnormal adenohypophysis morphology / MGI
- decreased renal glomerulus number / MGI
- decreased oocyte number / MGI
- abnormal female germ cell morphology / MGI
- absent Mullerian ducts / MGI
- decreased somatotroph cell number / MGI
- decreased gonadotroph cell number / MGI
- decreased thyrotroph cell number / MGI
- oocyte degeneration / MGI
- abnormal metanephric mesenchyme morphology / MGI
- abnormal nephrogenic mesenchyme morphogenesis / MGI
- postnatal lethality, complete penetrance / MGI
- neonatal lethality, complete penetrance / MGI
- small metanephros / MGI
B6;129P2-Wnt4tm2(EGFP/cre)Svo/Oulu
Status | Available to order |
EMMA ID | EM:10011 |
International strain name | B6;129P2-Wnt4tm2(EGFP/cre)Svo/Oulu |
Alternative name | Wnt-4-Cre |
Strain type | Targeted Mutant Strains : Knock-in |
Allele/Transgene symbol | Wnt4tm2(EGFP/cre)Svo |
Gene/Transgene symbol | Wnt4 |
Information from provider
Provider | Jingdong Shan |
Provider affiliation | Department of Biochemistry and Molecular Medicine, University of Oulu |
Additional owner | Dr. Seppo Vainio, University of Oulu, Oulu, Finland |
Genetic information | Targeted construct in 129/Ola ES cells replaces the Wnt4 gene after the promoter with cre recombinase. |
Phenotypic information | Homozygous:Wnt4-cre homozygotes have the same phenotype as Wnt4 knockouts. Kidney malformation and female to male sex reversal. Homozygotes die few days after birth.Heterozygous:Heterozygous mice have no reported phenotypes. |
Breeding history | Targeted mutation was directly maintained on a C57BL/6 background. |
References |
|
Homozygous fertile | not known |
Homozygous viable | no |
Homozygous matings required | no |
Immunocompromised | no |
Information from EMMA
Archiving centre | University of Oulu, Oulu, Finland |
Animals used for archiving | heterozygous C57BL/6JOlaHsd, wild-type C57BL/6NCrl |
Stage of embryos | 2-cell |
Disease and phenotype information
MGI phenotypes (gene matching)
Literature references
- Generation of an allele to inactivate Wnt4 gene function conditionally in the mouse.;Shan Jingdong, Jokela Tiina, Peltoketo Hellevi, Vainio Seppo, ;2009;Genesis (New York, N.Y. : 2000);47;782-8; 19830824
- Mapping of the fate of cell lineages generated from cells that express the Wnt4 gene by time-lapse during kidney development.;Shan Jingdong, Jokela Tiina, Skovorodkin Ilya, Vainio Seppo, ;2010;Differentiation; research in biological diversity;79;57-64; 19740593
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